CD146 (MCAM) in human cs-DLK1−/cs-CD34+ adipose stromal/progenitor cells
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چکیده
منابع مشابه
Melanoma-associated adhesion molecule MUC18/MCAM (CD146) and transcriptional regulator mader in normal human CNS.
The proteins MUC18 and Mader have been identified as markers of tumor progression in melanoma cells. MUC18, also known as MCAM (melanoma cell adhesion molecule) and as CD146 (endothelial antigen), is a cell adhesion molecule belonging to the immunoglobulin superfamily. Mader is a transcriptional regulator shown to negatively regulate EGR-1. As it is known that neoplastic cells of neuroectoderma...
متن کاملReceptor Lymphocyte and Is an Endothelial Adhesion Promotes Rolling via Microvilli Induction in Endothelium Interaction: MCAM/CD146 Molecule (MCAM)/CD146 in Lymphocyte Dual Role of Melanoma Cell Adhesion
متن کامل
Lymphocyte and Is an Endothelial Adhesion Promotes Rolling via Microvilli Induction in Endothelium Interaction: MCAM/CD146 Molecule (MCAM)/CD146 in Lymphocyte Dual Role of Melanoma Cell Adhesion
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A Novel mAb against a Human CD34 Peptide Reacts with the Native Protein on CD34+ Cells
Background: Human CD34 is a transmembrane glycoprotein which is expressed in human hematopoietic stem cells (HSCs) and the small- vessel endothelial cells of a variety of tissues. CD34 plays a critical role as a marker for diagnosis and classification of leukemia. Anti CD34 antibodies are used for isolation and purification of HSCs from bone marrow, peripheral blood and cord blood. Objective: ...
متن کاملParticipation of CD34-enriched mouse adipose cells in hair morphogenesis.
Adipose-derived stromal vascular fraction (SVF) cells are heterogeneous in nature, containing a number of different cell types. Recent studies indicate that CD34 may be a specific marker for adipose-derived mesenchymal stem cells (ADMSCs). Using their participation in hair morphogenesis as a model, the multi-differentiation potential of adult stem cells was investigated. In addition, adipose ti...
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ژورنال
عنوان ژورنال: Stem Cell Research
سال: 2017
ISSN: 1873-5061
DOI: 10.1016/j.scr.2017.05.004